Michele 
Member since May 4, 2014

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Re: “The Scary Man Lady

But maybe some of those words are too "big" for you to "understand."

5 likes, 0 dislikes
Posted by Michele on 05/04/2014 at 6:49 PM

Re: “The Scary Man Lady

Daniel, perhaps you should read up on science.

Typically, every fetus derives one chromosome from the mother ‘X,’ and a second sex chromosome from the father, 'X' or "Y.' Typically, a fetus having one X and one Y chromosome will develop as male because genes on the ‘Y’ chromosome play a vital role in triggering the hormones which masculinise the fetus, ensuring that his brain and sex organs are congruent. Conversely, typically, a fetus having two X chromosomes will develop as a female so that her brain and sex organs are congruent with each other. The way that a fetus develops in terms of sex and gender development are in a large part related, but not entirely, so that typically, an XY baby, showing the external characteristics consistent with a male will grow to adulthood identifying himself as a man and an XX baby, showing the external characteristics consistent with a female will typically grow up comfortably identifying herself as a woman.
While this scenario applies to most of us, it does NOT apply to all. For most of us, despite considerable variances, we are close enough to one end or the other of the gender/sex spectrum that we have a solid gender identity and do not question out sex/gender. Since this is true for most, it is assumed to be true for all babies. NOT TRUE.

Perhaps this example will make the sex/gender spectrum easier to understand before we move on to the "scary man lady" concept.

Someone with complete Androgen Insensitivity Syndrome will have XY chromosomes. It would be expected that the fetus would develop typically into a boy, and would have a penis and testicles, externally, rather than a vagina and clitoris, uterus, etc. With cAIS, despite the presence of the ‘Y’ chromosome, the fetus is partially insensitive to the androgens (male hormones) and therefore is not masculinised. This results in a mixture of female and male characteristics: the baby is born with the external appearance of a girl and retains female brain characteristics; she grows up identifying herself as a woman. It may be only at puberty, when the failure to menstruate is apparent, that the underlying condition is diagnosed. This XY female has no uterus, often a shortened vagina, or none, and undescended testes.

Now to move on to the "scary man lady" concept of gender variance. I am not going to discuss Mo specifically, because it is not my place and because I do not know them well enough to feel comfortable that I am identifying them in the most accurate way. I am going to talk about gender variance in general. I have seen it firsthand. My brother was born biologically as my sister. But HE is most certainly my brother.

Gender variance is becoming more and more understood in the scientific and medical worlds as biological. The most current medical viewpoint is that gender variance is associated with non-typical neurodevelopment of the brain at the fetal stage. Small areas of the brain that are distinctly different between males and females in the general population are also different in individuals who are experiencing gender variance, as in these areas of the brain have developed in opposition of the given sex and are congruent with the sex that person identifies with. See (1) and (2) at the end of this commentary for the studies. One study on adult MtF transgender individuals found that the hypothalmac networks had developed in such a way as to react as builogical women, not as biological men, when presented in oderous steroids to smell, “Because our MFTRs were nonhomosexual, the results are unlikely to be an effect of sexual practice. Instead, the data implicate that transsexuality may be associated with sex-atypical physiological responses in specific hypothalamic circuits, possibly as a consequence of a variant neuronal differentiation.”

In addition, there have been a number of twin studies conducted that show that gender nonconformity is heritable. One of them is referenced at (3).

(1) Zhou J-N, Swaab DF, Gooren LJ & Hofman MA. (1995) Sex Difference in the Human Brain and its Relation to Transsexuality. Nature 378, 68-70. “…in one of the human brain structures that is different between men and women, a totally female pattern was encountered in six male to female transsexual (people)…This was not due to cross-sex hormone treatment. These findings show that a biological structure in the brain distinguishes male to female transsexuals from men”; Cited by Gooren LJ, University Hospital, Vrije Universiteit of Amsterdam, affidavit in Bellinger v Bellinger, TLR 22-11-2000.

(2) Kruijver FPM, Zhou J-N, Pool CW, Hofman MA, Gooren LJG and Swaab DF. (2000) Male to female transsexuals have female neuron numbers in a limbic nucleus. The Journal of Clinical Endocrinology & Metabolism 85(5):2034-2041. “Regardless of sexual orientation, men had almost twice as many somatostatin neurons as women. The number of neurons in...male to female transsexuals was similar to that of the females…In contrast, the neuron number of female to male transsexuals was found to be in the male range…The present findings of somatostatin neuronal sex differences in the BSTc (a part of the brain) and its sex reversal in the transsexual brain clearly support the paradigm that in transsexuals sexual differentiation of the brain and genitals may go into opposite directions and point to a neurobiological basis of gender identity disorder“.

(3) Coolidge, F; Thede, L; Young, S (4 April 2002). "The Heritability of Gender Identity Disorder in a Child and Adolescent Twin Sample". Behavior Genetics 32 (4): 251–257.
“Overall, the results support the hypothesis that there is a strong heritable component to GID. The findings may also imply that gender identity may be much less a matter of choice and much more a matter of biology.”

(4) Berglund, H.; Lindström, P.; Dhejne-Helmy, C.; Savic, I. (2007). "Male-to-Female Transsexuals Show Sex-Atypical Hypothalamus Activation When Smelling Odorous Steroids". Cerebral Cortex 18 (8): 1900–1908.

10 likes, 2 dislikes
Posted by Michele Grossman on 05/04/2014 at 4:05 PM

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